Astrocytes and Microglia Drive Alzheimer's Progression More Than Neurons
New single-nucleus and spatial transcriptomics data reveal glial cells—not just neurons—are central players in Alzheimer's disease pathology.
Cognitive performance, neuroplasticity, dementia prevention, and brain aging research
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New single-nucleus and spatial transcriptomics data reveal glial cells—not just neurons—are central players in Alzheimer's disease pathology.
A randomized pilot study finds PEMF therapy significantly improved fatigue, sleep, depression, and walking capacity in post-COVID-19 patients over 5 weeks.
A landmark review reveals the ANS regulates metabolism, immunity, and aging — far beyond simple fight-or-flight control.
A post hoc analysis of the DISTAL trial finds EVT plus medical therapy significantly improves brain tissue preservation in hard-to-reach vessel occlusions.
Researchers propose redesigning memory clinic services to match patient complexity with the right level of specialist oversight.
A novel brain organoid stress model reveals how combining walnut peptide and theanine activates BDNF pathways to reduce anxiety and boost cognition.
New research reveals the brain actively directs development of its surrounding lymphatic immune network via specialized glial cells and neural signals.
A landmark review maps three distinct pathways linking brain and heart function, with major implications for neurological and cardiovascular disease.
Recent NIA findings spotlight lifestyle factors cutting Alzheimer's risk by 60%, calorie restriction slowing aging, and a gene regulating plaque toxicity.
Domine a arquitetura molecular dos sistemas glinfático e linfático meníngeo — dos mecanismos de regulação do AQP4 à terapia com VEGF-C — e explore as intervenções de ponta que atuam sobre essas vias para a prevenção de doenças neurodegenerativas.
The National Institute on Aging outlines recent priorities in aging and Alzheimer's disease research, including budget planning and training initiatives.
A Cochrane review of two RCTs finds very low-certainty evidence that IGF-1 neither prevents nor worsens retinopathy of prematurity in extremely preterm infants.