Excessive Daytime Napping Linked to Higher Mortality Risk in Older Adults
New JAMA research finds that too much daytime napping in older adults may signal or accelerate mortality risk.
Sleep optimization, mindfulness practices, stress reduction, and their impact on longevity
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New JAMA research finds that too much daytime napping in older adults may signal or accelerate mortality risk.
New research argues that nightmares are an underrecognized suicide risk factor requiring broader clinical intervention than imagery rehearsal therapy alone.
A sweeping analysis of U.S. university students from 2000–2023 reveals worsening sleep sufficiency rates over more than two decades.
A completed trial tests whether internet-based CBT-I can reduce both insomnia severity and alcohol consumption in heavy-drinking adults.
New research reveals how sleep-like brain waves coordinate memory replay and flexible thinking in real time.
New research maps beat-by-beat BP spikes following each apnea episode, revealing cardiovascular stress invisible to standard sleep studies.
A large UK Biobank study finds sleep duration, napping, and sleeplessness independently predict white matter damage tied to dementia risk.
New research maps a stress-signaling cascade linking p53 activation to brain inflammation and depression, with curcumin interrupting it.
Multi-night home sleep tests rival lab polysomnography for uncomplicated OSA, cutting costs and improving patient experience.
Domine os mecanismos moleculares da depuração cerebral dependente do sono, disseque o papel cronobiológico da melatonina e aplique protocolos baseados em evidências — da TCC-I a auxiliares farmacológicos — para desenvolver um sono restaurador voltado à longevidade.
Explore as mudanças mecanísticas na arquitetura do sono com o envelhecimento — da redução do sono de ondas lentas aos relógios circadianos desregulados — e como essas alterações desencadeiam disfunções hormonais, inflamatórias e metabólicas.
Mergulhe na maquinaria molecular por trás da saúde cerebral ao longo da vida — das cascatas de sinalização BDNF-TrkB e da neurogênese hipocampal adulta à depuração glinfática de amiloide e intervenções de ponta, incluindo senolíticos, agonistas de GLP-1 e estimulação transcraniana.