Parkinson's disease, dopamine pathways, movement disorders, and neuroprotection
125 Artikel
Forscher der Universität Cambridge haben ein Gennetzwerk entdeckt, das die Nervenreparatur abschaltet – und ein bereits bestehendes Hormonmedikament, das es wieder aktiviert.
Ein neu identifizierter Proteasom-Aktivator, PA200, beseitigt toxische Alpha-Synuclein-Aggregate und erhält seine Funktion selbst dann aufrecht, wenn die standardmäßigen Proteinabbausysteme versagen.
Forscher reagieren auf Kritik an ihrer Studie, die zeigt, dass eine Überdruckbeatmungstherapie kognitive Ergebnisse bei Parkinson-Erkrankungen verbessern könnte.
A completed RCT tests whether 6 months of moderate aerobic exercise protects memory and brain structure in older adults at genetic risk for Alzheimer's.
A professional oboist discovered that playing her instrument or singing completely suppressed her debilitating blepharospasm symptoms.
Specific AQP4 genetic variants alter glymphatic drainage efficiency, directly influencing motor symptom severity and progression in early Parkinson's disease.
A major review reveals how the glymphatic system's failure drives neurodegeneration and brain tumor resistance — and how to fix it.
New mouse study reveals how the brain's waste-clearance system modulates toxic protein buildup linked to Parkinson's disease.
New research links age at Parkinson's onset and family history to the likelihood of carrying disease-causing genetic variants.
β-endorphin released during exercise wraps around toxic Aβ aggregates, forming a 'corona' that shields brain cells from Alzheimer's damage.
From Alzheimer's to Parkinson's, a wave of novel FDA approvals is expanding treatment options for serious neurological conditions.
A 79-year-old man developed involuntary movements weeks after carotid surgery, revealing a rare stroke complication.
