20 articles
A rigorous MRI-based study finds that repeated hypoglycemia episodes leave brain glucose transport kinetics unchanged, challenging a leading hypothesis.
New research reveals how adrenaline from hypoglycemia causes prolonged inflammatory responses lasting up to seven days.
New research reveals hypoglycemia after glucose tolerance tests in some type 2 diabetes patients with higher insulin sensitivity.
New research validates high-throughput methods for studying mitochondrial dysfunction in malignant hyperthermia susceptibility.
New research reveals specific brain cells critical for maintaining body temperature when food is restricted, with major implications for fasting safety.
UCLA researchers outline best practices for respirometry, the gold-standard method for measuring cellular energy and mitochondrial function.
New research reveals pulsed electromagnetic field therapy selectively enhances ATP-linked mitochondrial respiration, pointing to a specific cellular energy mechanism.
New research reveals fatty liver disease follows circadian patterns linked to insulin resistance and reduced nighttime insulin availability.
Restoring mitochondrial complex I function in fat cells prevents obesity-related metabolic dysfunction, even without weight loss.
Ketone body ฮฒ-OHB prevents sepsis-induced liver damage by modifying antioxidant enzyme SOD2, offering new therapeutic approach.
New research reveals pulmonary arterial hypertension involves systemic metabolic dysfunction across multiple organs and immune cells.
New research reveals how obesity creates a cascade of cardiovascular and metabolic dysfunction that severely limits exercise capacity.
New research reveals mitochondria use lactate to prevent oxidative damage rather than generate energy, challenging conventional wisdom.
Knocking out BNIP3 in macrophages reduced adipose inflammation and improved insulin sensitivity in obese mice, revealing a new therapeutic target.
New study shows fasted morning resistance training produces more consistent glucose responses than fed afternoon sessions.
New research reveals how obesity during pregnancy triggers genetic changes that cause lasting muscle dysfunction in offspring.
Low-intensity resistance training with blood flow restriction improved muscle strength and mitochondrial function better than conventional exercise.
Researchers discover how disrupted circadian rhythms in fat cells impair mitochondrial function, leading to metabolic syndrome.
Machine learning clusters CGM data into four distinct glycemic patterns, each linked to unique diabetic complication risks.
New evidence reveals mitochondria form functionally distinct subpopulations within tissues and even single cells, reshaping how we understand energy metabolism.
