Gut Bacteria Functions Change With Age and Drive Metabolic Disease Risk
Large study reveals how gut microbe proteins shift with aging and contribute to diabetes and metabolic disorders.
20 articles
Large study reveals how gut microbe proteins shift with aging and contribute to diabetes and metabolic disorders.
A comprehensive review reveals how obesity and aging converge through shared inflammatory biomarkers, accelerating disease risk in the elderly.
Large NHANES study reveals metabolic syndrome causes measurable acceleration in epigenetic aging clocks, offering new targets for intervention.
Multi-omic analysis reveals GLP-1 receptor agonists counter aging processes across multiple organ systems and biological pathways.
A 6-month AI-driven personalized diet intervention cut BMI by 33% and reduced biological age by 8 years in morbidly obese patients.
New review reveals how saturated fat-rich diets trigger neuroinflammation and cognitive decline, but offers actionable prevention strategies.
A noninvasive 13-variable score reveals how fast your liver is aging โ and accelerated liver aging raises mortality risk by up to 85%.
Mice lacking the HDAC9 gene gained less fat with age, accumulated fewer senescent cells, and showed improved mitochondrial respiration in adipose tissue.
Second-generation epigenetic clocks show strong associations with metabolic syndrome and type 2 diabetes risk prediction.
Scientists discover breath analysis can instantly detect gut bacteria composition, opening new paths for microbiome diagnostics.
Researchers analyzed 2,911 proteins in 50,506 people to create a frailty score that predicts disease risk and biological aging.
Diabetes drugs like empagliflozin show surprising anti-aging effects โ mimicking caloric restriction and reducing senescent cell burden.
New study shows DunedinPACE aging biomarker can predict who will develop metabolic syndrome years in advance, outperforming other epigenetic clocks.
A new review links feline age-related obesity to SASP-driven chronic inflammation, offering translational insights for human metabolic disease.
New cryo-EM structures reveal how LYCHOS protein senses cholesterol levels to regulate mTORC1 signaling, offering drug targets for aging.
New research reveals GLP-1 medications like semaglutide may reduce muscle strength in older adults, even when muscle mass appears preserved.
Flavo-Proxylane topical treatment plus ultrasound reduced skin laxity by 44% in people using weight loss medications.
Transcriptomic analysis of Pseudomonas nitroreducens TX1 identifies 241 differentially expressed genes during surfactant degradation.
A newly identified mechanism shows how omega-3 fatty acids protect aging kidneys โ and why low FFAR4 receptor expression accelerates kidney disease.
New evidence reveals mitochondria form functionally distinct subpopulations within tissues and even single cells, reshaping how we understand energy metabolism.