20 articles
Stanford researchers found ribosome stalling in aging brain cells triggers faulty proteins and toxic clumps linked to Alzheimer's disease.
The SenNet atlas reveals where aging cells accumulate in human tissues, opening new doors for senolytic therapies and healthspan extension.
New research identifies FTL1 protein as key driver of brain aging. Reducing it restored memory and neural connections in mice.
Declining Menin in the hypothalamus drives inflammation and aging. Restoring it reversed decline; D-serine amino acid improved cognition in mice.
New p-tau217 blood test forecasts Alzheimer's symptoms within 3-4 years, potentially revolutionizing early intervention strategies.
Buck Institute finds the longevity-linked APOE2 variant shields neurons from DNA damage and cellular senescence, explaining its Alzheimer's protection.
Scientists discover that subtle changes in blood protein structure can identify Alzheimer's earlier than current tests.
Recent NIA findings spotlight lifestyle factors cutting Alzheimer's risk by 60%, calorie restriction slowing aging, and a gene regulating plaque toxicity.
Raising Sox9 levels in aging astrocytes reduced amyloid plaque buildup and protected memory in mouse models with existing Alzheimer's symptoms.
A 3-year UT Dallas study of nearly 4,000 adults found measurable brain health gains at every age — even in your 80s and 90s.
UCLA scientists discover why some neurons resist Alzheimer's better than others, revealing a natural protein cleanup system that could lead to new treatments.
Scientists discover tanycytes help clear toxic tau from the brain. When damaged, these cells may allow Alzheimer's progression.
Scientists find that TDP43 protein regulates DNA repair systems, linking neurodegeneration to cancer when repair goes wrong.
A 17-country study finds U.S. midlife health is declining across generations while peer nations improve — here's why it matters.
Researchers captured live footage of protein clumping in Alzheimer's disease and discovered how to potentially reverse the damage.
New AI system reveals how genes control each other in Alzheimer's brains, uncovering potential treatment targets.
A new tool that boosts mitochondrial activity in neurons restored memory in dementia mice, suggesting energy failure drives Alzheimer's symptoms.
UC Riverside researchers show amyloid beta may displace tau from neurons, triggering brain cell breakdown before plaques even form.
Blocking the protein PTP1B boosted memory and cleared brain plaque in mice, with potential links to diabetes and obesity treatment.
Speed-of-processing training in adults 65+ showed lasting protection against dementia in groundbreaking 20-year study.
