20 articles
New technique maps thousands of neural connections simultaneously, potentially revolutionizing understanding of brain aging and disease.
New AI system reveals how genes control each other in Alzheimer's brains, uncovering potential treatment targets.
Researchers identified the gene that transports queuosine, a micronutrient crucial for brain health and cancer defense, solving a decades-old mystery.
The SenNet atlas reveals where aging cells accumulate in human tissues, opening new doors for senolytic therapies and healthspan extension.
McGill researchers pinpointed two brain cell types altered in depression, opening doors to targeted biological treatments.
MIT researchers discovered how a schizophrenia gene mutation prevents brain adaptation to new information and found a way to restore normal function.
Stanford researchers found ribosome stalling in aging brain cells triggers faulty proteins and toxic clumps linked to Alzheimer's disease.
Raising Sox9 levels in aging astrocytes reduced amyloid plaque buildup and protected memory in mouse models with existing Alzheimer's symptoms.
Scientists discover that subtle changes in blood protein structure can identify Alzheimer's earlier than current tests.
New NIH research reveals the brain-cell signaling mechanism behind GLP-1 drug plateaus and a potential way to extend their effects.
Scientists find that TDP43 protein regulates DNA repair systems, linking neurodegeneration to cancer when repair goes wrong.
A molecule called OLE restores microglia's protective function, reducing amyloid plaques and improving memory in animal models.
MIT researchers discovered that 30% of adult brain synapses are dormant but ready to activate, reshaping how we understand lifelong learning.
Scientists identify how nitric oxide triggers cellular changes in autism, offering hope for targeted therapies.
New mouse research finds the trendy D+Q senolytic combo severely damages myelin, raising red flags for longevity self-experimenters.
New research reveals intelligence comes from how efficiently brain networks communicate together, not from one 'smart' brain area.
Buck Institute finds the longevity-linked APOE2 variant shields neurons from DNA damage and cellular senescence, explaining its Alzheimer's protection.
Researchers turned ordinary brain cells into plaque-clearing machines, reducing Alzheimer's amyloid by half in mice with one treatment.
New p-tau217 blood test forecasts Alzheimer's symptoms within 3-4 years, potentially revolutionizing early intervention strategies.
Researchers restored a key brain circuit in mice and reversed anxiety and social withdrawal, pointing to a powerful new therapeutic target.
