Scientists Switch On Nerve Regeneration Once Thought Lost Forever
Cambridge researchers found a gene network that shuts off nerve repair โ and an existing hormone drug that reactivates it.
20 articles
Cambridge researchers found a gene network that shuts off nerve repair โ and an existing hormone drug that reactivates it.
Researchers turned ordinary brain cells into plaque-clearing machines, reducing Alzheimer's amyloid by half in mice with one treatment.
Scientists used red blood cells to transport healthy mitochondria into diseased brain cells, showing promise for Parkinson's treatment.
The SenNet atlas reveals where aging cells accumulate in human tissues, opening new doors for senolytic therapies and healthspan extension.
A molecule called OLE restores microglia's protective function, reducing amyloid plaques and improving memory in animal models.
Researchers discover TMEM175, a cellular safeguard that prevents toxic buildup linked to Parkinson's when it malfunctions.
Stanford researchers found ribosome stalling in aging brain cells triggers faulty proteins and toxic clumps linked to Alzheimer's disease.
Scientists discover tanycytes help clear toxic tau from the brain. When damaged, these cells may allow Alzheimer's progression.
New technique maps thousands of neural connections simultaneously, potentially revolutionizing understanding of brain aging and disease.
UCLA scientists discover why some neurons resist Alzheimer's better than others, revealing a natural protein cleanup system that could lead to new treatments.
Scientists discover that subtle changes in blood protein structure can identify Alzheimer's earlier than current tests.
Blocking the protein PTP1B boosted memory and cleared brain plaque in mice, with potential links to diabetes and obesity treatment.
Astrocytes, once thought passive, actively control fear memories in the brain, opening new paths for anxiety disorder treatments.
New research reveals depression may stem from cellular energy imbalances, offering hope for earlier diagnosis and targeted treatments.
Baylor scientists find tubulin redirects rogue brain proteins away from toxic aggregates, pointing to a novel neurodegenerative disease strategy.
Raising Sox9 levels in aging astrocytes reduced amyloid plaque buildup and protected memory in mouse models with existing Alzheimer's symptoms.
Speed-of-processing training in adults 65+ showed lasting protection against dementia in groundbreaking 20-year study.
Researchers captured live footage of protein clumping in Alzheimer's disease and discovered how to potentially reverse the damage.
New p-tau217 blood test forecasts Alzheimer's symptoms within 3-4 years, potentially revolutionizing early intervention strategies.
McGill researchers pinpointed two brain cell types altered in depression, opening doors to targeted biological treatments.